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From a research perspective this validates the angiogenic mechanism of GHK-Cu, similar to BPC-157 and TB-500

aeruginosa T3SS is induced by several conditions including low Ca 2+ and host cell contact through ExsA [[35], [36], [37]], the redox control of T3SS via Vfr could be another layer of modulation where oxidizing condition could dampen T3SS induction

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