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Endogenous GH is secreted in discrete pulses, primarily during sleep and in response to physiological stimuli, and this pulsatile pattern is understood to be important for the downstream biological effects of GH, including its influence on IGF-1 production, body composition, and tissue repair processes

Under ferroptotic stress, JNK activation leads to phosphorylation of Beclin-1 at Ser90/93, strengthening its interaction with the VPS34 complex and stimulating autophagosome formation (58,59)