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In light of this, the pathophysiological relevance of the ASPL-VCP interaction warrants further investigation in more physiologically relevant systems, such as patient-derived neurons, animal models, or chronic stress paradigms, which may better recapitulate the characteristic pathological accumulations of ubiquitin and SQSTM1 observed in MSP and related disorders

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Glutamate can be further catabolized through the TCA cycle (via conversion to KG) or serve as a substrate for glutathione synthesis