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These metabolic changes lead to glomerular hyperfiltration, enlargement, and increased proximal tubular glucose reabsorption via sodium-glucose cotransporter 2, resulting in elevated intraglomerular pressure and hyperfiltration [25]

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First, accumulating evidence has shown that the expression and function of key efferocytosis receptors, MerTK (183), LRP1 (184), and SR-BI (185) are impaired in advanced atherosclerosis

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The BSO-induced increase in glutamate was accompanied by an increase in mEPSC frequency (Fig