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thus, in one way, glutathione regulates -GCS activity via a negative feedback mechanism [13]

As such, hepatic steatosis, secondary to compromised triglyceride export, is common in patients with genetic defects in the apoB or MTTP gene (hypobetalipoproteinemia and abetaproteinemia, respectively) [112, 113]

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At The Center for Occupational and Environmental Medicine (COEM), intravenous administration is considered when laboratory findings, exposure history, or clinical presentation suggest increased oxidative burden or impaired detox capacity