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Monoacylglycerol binds to GPR119 in L cells to promote the release of GLP-1, GIP and insulin ( 2+ were observed following GPBA-R activation in single GLUTag cell, and bile acid and GPBA-RA increased GLP-1 secretion in intestinal cultures (Parker et al., 2012b)

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[DOI] [PubMed] [Google Scholar] 29.Santos M.A.T.D

reported that glucagon-induced insulin secretion was inhibited by Exendin 9-39 in purified rat beta-cells ( Nevertheless, the propensity for glucagon to augment insulin secretion through the beta-cell GLP-1R adds another layer of complexity to characterizing the physiological importance of alpha-cell-derived GLP-1 to the incretin effect