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By investigating the mechanisms through which radiation increases cellular reactive oxygen species (ROS) and initiates ferroptosis, we aim to develop targeted therapeutic strategies that maximize treatment efficacy and minimize neurotoxicity

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10, 11 Moreover, the pharmacological antioxidant N -acetylcystein (NAC) enhances T-cell function and proliferation by inhibition of nuclear factor- B (NF- B)

doi:10.1056/NEJMoa2032183 Jastreboff AM, Aronne LJ, Ahmad NN, et al